Tomohiro Ide
University of Hyogo(JP)Hyogo University(JP)
Publications by Year
Research Areas
Peroxisome Proliferator-Activated Receptors, Adipose Tissue and Metabolism, Metabolism, Diabetes, and Cancer, Cholesterol and Lipid Metabolism, Adipokines, Inflammation, and Metabolic Diseases
Most-Cited Works
- → Targeted disruption of AdipoR1 and AdipoR2 causes abrogation of adiponectin binding and metabolic actions(2007)1,380 cited
- → Overexpression of Monocyte Chemoattractant Protein-1 in Adipose Tissues Causes Macrophage Recruitment and Insulin Resistance(2006)895 cited
- → The Mechanisms by Which Both Heterozygous Peroxisome Proliferator-activated Receptor γ (PPARγ) Deficiency and PPARγ Agonist Improve Insulin Resistance(2001)636 cited
- → Impaired Insulin Signaling in Endothelial Cells Reduces Insulin-Induced Glucose Uptake by Skeletal Muscle(2011)428 cited
- → Polyunsaturated Fatty Acids Suppress Sterol Regulatory Element-binding Protein 1c Promoter Activity by Inhibition of Liver X Receptor (LXR) Binding to LXR Response Elements(2002)418 cited
- → Absence of Sterol Regulatory Element-binding Protein-1 (SREBP-1) Ameliorates Fatty Livers but Not Obesity or Insulin Resistance in Lep/Lep Mice(2002)365 cited
- → SREBPs suppress IRS-2-mediated insulin signalling in the liver(2004)315 cited
- → Cross-Talk between Peroxisome Proliferator-Activated Receptor (PPAR) α and Liver X Receptor (LXR) in Nutritional Regulation of Fatty Acid Metabolism. I. PPARs Suppress Sterol Regulatory Element Binding Protein-1c Promoter through Inhibition of LXR Signaling(2003)293 cited
- → Inhibition of RXR and PPARγ ameliorates diet-induced obesity and type 2 diabetes(2001)292 cited
- → p53 Activation in Adipocytes of Obese Mice(2003)215 cited