Andreas Markl
Publications by Year
Research Areas
COVID-19 Clinical Research Studies, Cardiac Fibrosis and Remodeling, Cardiac Imaging and Diagnostics, COVID-19 epidemiological studies, Cardiovascular Function and Risk Factors
Most-Cited Works
- → Enhanced Expression of β3-Adrenoceptors in Cardiac Myocytes Attenuates Neurohormone-Induced Hypertrophic Remodeling Through Nitric Oxide Synthase(2013)144 cited
- → Cardiac myocyte β3-adrenergic receptors prevent myocardial fibrosis by modulating oxidant stress-dependent paracrine signaling(2017)75 cited
- → HMGCoA reductase inhibition reverses myocardial fibrosis and diastolic dysfunction through AMP-activated protein kinase activation in a mouse model of metabolic syndrome(2013)74 cited
- → Effects of Environmental Factors on Severity and Mortality of COVID-19(2021)54 cited
- → Insulin receptor substrate-1 and -2 mediate resistance to glucose-induced caspase-3 activation in human neuroblastoma cells(2011)17 cited
- → Concerto Pin(2012)6 cited
- Statins prevent LV remodelling through anti-fibrotic effects associated with AMPK activation rather than direct effects on cardiomyocytes in a mouse model of metabolic syndrome(2012)
- HMGCoA Reductase Inhibition Reverses Myocardial Fibrosis and Diastolic Dysfunction Through AMPK Activation in a Mouse Model of Metabolic Syndrome(2012)
- → 309Protection from cardiac fibrosis is induced by beta3 adrenergic receptor in cardiac myocyte through inhibition of paracrine signalling to fibroblast : proteomic analysis of the myocyte secretome(2014)